Recently it has been shown that high levels of CXCL10 are present in patients with autoimmune thyroiditis (AT), in particular in the presence of hypothyroidism [68], and an involvement of Th1 immune response in the induction of AT [86], Graves' disease, and Graves' ophthalmopathy [87] has been shown. This evidence concerns the gene CXCL10 and ataxia telangiectasia.