In a mouse model of atherosclerosis (i.e., angiotensin II infusion in ApoE −/−) Gil-Bernabe et al. demonstrated that the low-dose ethanol treatment group had fewer atheromatous lesions, and increased secretion of stromal cell-derived factor-1 (SDF-1) with subsequent enhanced mobilization of progenitor cells, compared to the no alcohol controls [67]. Here, CXCL12 is linked to atherosclerosis.