Notable in particular was expression of genes such as VIM, TGFß1, ZEB2, FOXC1, and CXCR4, associated with the induction and maintenance of EMT, a process by which epithelial cells transition to a more mesenchymal phenotype, both morphologically and biochemically [49]–[56], thereby increasing cell invasiveness and the link to cancer progression and poor prognosis [56], [57]. The gene discussed is VIM; the disease is cancer.