Psoriasis is characterized by the upregulation of the cytokines IFN-α, IFN-γ, TNF-α, IL-17, IL-22, and IL-23 [59], while TNF-α, IFN-γ, and Th17+ T cells have been shown to be important in HIV-1 controllers [60]–[62]. The gene discussed is IL23A; the disease is psoriasis.