Since MYH9 codes for non-muscle myosin heavy chain IIA and its expression correlated exceptionally well with AXL (expression profile correlation 0.74; Figure 2), we conjecture that AXL and MYH9 in myosin IIA may associate with each other and function together to confer movement to migrating tumor cells, particularly in the process of rear retraction, for which myosin IIA is required in several cell types [11] (Figure 12, interactions 34 and 35). The gene discussed is MYH9; the disease is neoplasm.