CALB1 and epilepsy: There were visibly fewer N200-, SMI32-, calretinin-, calbindin- and parvalbumin-immunopositive cells in the CA regions of Case 2 and the MTLE control without NDE1 deletion, than in Case 1 and non-epilepsy controls, in keeping with the presence of hippocampal sclerosis in the MTLE disease control and Case 2.