RAD51 and infection: The isolation of this group of genes must be related to the repair of Mu insertions and not to repair of random double strand breaks generated upon Mu infection, because (1) they are dependent on Mu integration (i.e. infection with MuAam1093 phage does not significantly affect the viability of the priA and recA hosts as compared to wild type; Figure S6A), and (2) Mu-induced mutations are known to be tightly linked to Mu i.e. they are not random [28].