Since both IL41I [19], and IFI30 [20], [21] participate in the down-regulation of Th1 mediated inflammation, a crucial element in atherosclerosis [22], [23], our results of high expression of IL41I and IFI30 may reflect a negative feedback response to Th1 mediated inflammation in the atherosclerotic process. This evidence concerns the gene IFI30 and atherosclerosis.