However, there is some indication, albeit a paucity of evidence, that the DNA repair capacity of LCLs from breast cancer samples is significantly lower than control subjects [56], that tumor-infiltrating Foxp3+ regulatory T cells can distinguish between high-risk breast cancer patients and those at risk of a late relapse [57] and that a fraction of eQTLs derived from the analysis of UK Adult Twin registry LCLs gene expression and genotype data overlap with those identified in a HapMap population [47]. The gene discussed is FOXP3; the disease is breast cancer.