However, the data showing that TNBC-TICs exhibit classical signs of aggressive phenotype shown by human TNBC including increased tumor growth (Figure 5) and enhanced metastatic potential (Figure 6), led us to hypothesize that the differential expression of mouse HspB1 and Hsp72/HspA1A, might be a possible explanation (Figure 7). The gene discussed is HSPB1; the disease is neoplasm.