In this study, we have confirmed that a high level of insulin resistance is a driving parameter in the pathophysiology of the dyslipidaemia seen in the MAO group compared to the MOA and LHO groups and found the typical lipid profile: increased plasma triglycerides, apoB-100, apoB-48, apoC-III and apoC-II, and decreased HDL-c and apoA-I. This evidence concerns the gene APOA1 and inherited lipid metabolism disorder.