Consistent with these observations, we found six genes, ADA, CHKA, DEGS2, OSBPL2, PPAPDC3, and Q5R387; which are involved in lipid biosynthesis, tumor cell growth by phosphatidyl-ethanolamine biosynthesis, negative regulation of myoblast differentiation and hydrolyzation of phospholipids into fatty acids etc. This finding is in agreement with Diamond et al.; that host cell lipid metabolism may represent an area for future HCV antiviral therapies [56]. The gene discussed is DEGS2; the disease is neoplasm.