Amongst the other “most plausible” risk genes worthy of mention are CD36 (aTOH4) and PSIP1 (cTOH7), both of which have plausible roles in lung function development, and genetic alterations (especially in the oncogene PSIP1) may well lead to NSCLC development (detailed in Table S4). This evidence concerns the gene CD36 and non-small cell lung carcinoma.