However, the biologically plausible hypothesis that aflatoxin exposure may induce chronic liver damage, which increases the susceptibility to schistosomiasis-associated morbidity—or vice versa, that the liver damage due to schistosomiasis infection may alter the metabolism of aflatoxin and also the synthesis of albumin, which could affect the AF-alb level—merits further analysis in studies designed to address this question. This evidence concerns the gene ALB and schistosomiasis.