Although the etiology and pathogenesis of FUS are not fully understood, viral infections and autoimmunity have been presumed to be involved in its development, as supported by the demonstration of local intraocular antiviral antibody production, increased expression of interferon-gamma (IFN-γ) and interleukin 6 in Fuchs’ patients [23-25]. Here, IL6 is linked to Autoimmunity.