In primary (CD127+132−: 18% T(E)M, CD127+132+: 25%, CD127−132+:27%; p = 0.13) and chronic HIV infection T(E)M cells were more evenly spread (CD127+132−: 18% T(E)M, CD127+132+: 25%, CD127−132+:35%; p = 0.06; Figure 1a)iii and b)iii). This evidence concerns the gene IL7R and HIV infectious disease.