In contrast, a mouse carrying a heterozygous 2.3 Mb deletion of MMU10, syntenic to the human region located between the PRMT2 and PDXK genes, showed similarities to Monosomy 21-associated intellectual disability (with impairments in learning and memory), as did a mouse carrying a heterozygous deletion of a 1.1 Mb segment on MMU17, syntenic to the human region located between the ABCG1 and RRP1B genes [14]. The gene discussed is PRMT2; the disease is Intellectual disability.