NAT2 and tuberculosis: These candidates include SYNJ2, which is involved in the phosphatidylinositol signaling pathway, and is upregulated after exposure to botulinum neurotoxins in SH-SY5Y cells [23], NAT2, which is involved in the metabolism of drugs used to treat tuberculosis [24], and AIMP1, which encodes a protein that is involved in the control of angiogenesis, inflammation, wound healing, and glucose homeostasis [25].