MLH1 and colorectal cancer: Qi et al. [184] examined methylseleninic acid (MSA, 0-10 μM), methyl selenocysteine (MSC, 0-500 μM), and sodium selenite (0-20 μM) in mismatch repair (MMR)-deficient HCT116 colorectal cancer cells and MMR-proficient HCT116 cells with MutL homolog 1 (MLH1) complementation.