EAU in B10.RIII mice induced by IRBP161–180, the best-studied model of uveitis recently shown to be IL-17 driven [23,44,55], has commonly been described as a monophasic disease [56], with a clinical peak about 2 weeks after immunization [37,57,58], then followed by remission and tolerance to reinduction; namely, EAU is a self-limited disease [1]. The gene discussed is IL17A; the disease is uveitis.