COL26A1 and polyp: Although the association signals detected in the analyses of both AERD and ATA subgroups were not deemed significant after multiple comparisons, four EMID2 variations (rs6949799, rs4727494, rs13233066, and EMID2_BL2_ht2) showing nominal association signals with polyp development in the AERD group in the current study was also marginally implicated in the risk of AERD in a Korean population using various modes of genetic inheritance [3], except for EMID2_BL2_ht2 which remained significant after multiple testing corrections in the previous report.