Quantitative analyses (Figure 3) of 1,000 randomly selected cancer cells by inForm software (Caliper) supported the imaging data in which both AR and E-cadherin expression was drastically decreased and activated c-Met signaling components led to EMT, such as elevated expression of c-Met, p-c-Met, p-NFκB p65, N-cadherin, vimentin, and RANKL were observed in LNCaP-RANKL, when compared to LNCaP-neo cells. The gene discussed is MET; the disease is cancer.