After tumor-induced expansion, they can be divided in two main subsets, depending primarily on their ancestors, but also on the suppression mechanisms they exert: monocytic MDSCs, with a CD11b+LY6G−LY6Chigh phenotype, and granulocytic MDSCs, with a CD11b+LY6G+LY6Clow phenotype. Here, ITGAM is linked to neoplasm.