BCL6 and Burkitt lymphoma: The authors proposed that hsa-miR-127 overexpression in EBV-positive BL may determine downregulation of PRDM1 and XBP-1, and the consequent persistence of BCL6 and germinal center phenotype in a B cell that is already differentiated in terms of surface immunoglobulin and mutation pattern toward a postgerminal center B cells.