Since T cells play an important role in the severity of cardiac damage in CVB3-induced myocarditis [31], we speculate that the observed decrease in cardiac apoptosis and subsequent viral load in CVB3+CAPs mice versus CVB3+PBS mice is due not only to the direct anti-apoptotic effects of CAPs, but also to their immunomodulatory features. The gene discussed is CAPS; the disease is myocarditis.