Since upregulation of RGC-32 expression has been linked to numerous cancers [31]–[34], potentially through the role of RGC-32 as an activator of the mitotic CDK1/cyclin B1 complex [27]–[28], these results may implicate RGC-32 upregulation in EBV-mediated tumourigenesis. This evidence concerns the gene CDK1 and cancer.