For example common functional variants in the gene for mannose binding lectin (MBL), a key complement-like constituent of innate immune defence, have been associated with susceptibility to KD,[28] and also coronary artery damage.[29] A role for MBL variants in susceptibility to certain infections, and to asthma and atopy has been reported, but data are inconsistent.[30] The available data suggest that KD is unlikely to be triggered by common allergens.[31]. Here, MBL2 is linked to asthma.