Despite the expected phenotypic heterogeneity, diffusion-weighted neuroimaging studies have yielded broadly consistent findings of dysconnectivity in limbic, thalamocortical and intracortical tracts in SZ and BD [5], [6], [7], [8], [9], [61], [62], [63], some of which parallel those in Sema6A mutants remarkably closely (this study and [40]). Here, SEMA6A is linked to Behcet disease.