Endotoxemia resulted in a cumulative GSTA1-1 excretion of 11.2 (6.2 to 13.0) μg compared with 5.1 (3.9 to 9.4) μg 12 hours after LPS administration in dipyridamole- and placebo-treated subjects, respectively. The gene discussed is GSTA1; the disease is serum lipopolysaccharide activity.