For example, alternative splicing in the neuronal α4 and α7 nAChR subunits is associated with Tourette syndrome [32] and schizophrenia [33], respectively, and that of the α1 and ε subunits of the muscle-type nAChR—with congenital myasthenic syndromes [34], [35], [36]. This evidence concerns the gene CHRNA7 and Tourette syndrome.