The beneficial effect of Ang-(1–7) infusion after myocardial infarction relies on the versatile bioactivity of the hormone, which comprises antihypertrophic, -fibrotic, and -thrombotic function, improvement of eNOS function, blockade of Ang-II-induced ROS production, and stimulation of endothelial-progenitor-cell-mediated angiogenesis [1, 2, 5–9]. Here, AGT is linked to myocardial infarction.