Differential expression of CYP1B1 in tumor cells creates an opportunity to develop potential strategies that may involve either inhibiting CYP1B1 activity (which as stated above activates procarcinogens) or using the metabolic activity of CYP1B1 to activate essentially non-toxic prodrugs (e.g., resveratrol and aryl oxime) to cytotoxic compounds. Here, CYP1B1 is linked to neoplasm.