IL4 and infection: Figure 4 shows that, in contrast to the protection afforded by conidia, SGG failed to confer resistance to infection and even favored fungal growth (Fig. 4A). No reduced inflammatory pathology was seen in the lung where actual fungal growth was observed in GpG+SGG-treated mice (Fig. 4B) and the cytokine pattern showed that SGG inhibited Ifnγ/Il10 and activated Il4 gene expression in the TLN, thus suggesting inhibition of protective Th1/Treg cells and promotion of Th2 responses (Fig. 4C).