In summary, the whole genome transcriptional analysis supports the notion that co-expression of the DSCAM and COL6A2 genes results in a transcriptional misregulation of genes involved in cell-cell adhesion and ECM-cell interaction, which may contribute to the observed increased adhesion of H9C2-DSCAM cells to collagen substrate, as well as genes that respond transcriptionally in patients with cardiomyopathies. The gene discussed is COL6A2; the disease is cardiomyopathy.