PTPRC and myelodysplastic syndrome: Assessment of H2A.X phosphorylation versus DNA content, illustrated in representative control and MDS samples in Figure 3, allowed analysis of p-H2A.X reactivity by cell cycle compartment (G0/G1, post-G1) in erythroid and myeloid progenitors using combinations of p-H2A.X reactivity, side scatter, CD45 antigen density, and DNA content.