A few reports exhibit the role of Tβ4 in the myocardial infarction settings where it promotes endothelial and myocardial cell survival, cardiac cell migration, activation of integrin linked kinase (ILK) and activation of Akt/Protein Kinase B that resulted in improved cardiac function and reduction in scarring [14], [16], [21]. Here, AKT1 is linked to myocardial infarction.