S100A8 and infection: Based on a series of experiments including the use of integrin β1 knockout cell lines (GD25 and GD25β1), gene silencing RNAs, function-blocking antibodies and competition experiments with a well-known integrin β1 bacterial adhesin (InvA from Yersinia), compelling evidence was provided that integrin β1 plays a crucial role for injection of CagA during infection of several non-polarised AGS and mouse cell lines [61].