Transgenic mice have often been used to investigate the physiopathology of muscular dystrophies [4-6]; however, the mutation remains in a murine context, and there are often major differences between humans and mice; for example, a mutation in the dystrophin gene results in a mild pathological phenotype in mdx mice but in a progressive and fatal disease (Duchenne muscular dystrophy; DMD) in humans. This evidence concerns the gene DMD and Duchenne muscular dystrophy.