Analysis of genes that were more highly expressed in the Type III-infected cells vs. Type II infections revealed significant overlap with gene sets associated with cell proliferation or oncogenic transformation (Table 1), whereas analysis of genes more highly expressed in Type II-infections identified gene sets up-regulated in response to TNF-signaling, interferon-stimulation, and NF-κB-stimulation (Table 2). The gene discussed is NFKB1; the disease is infection.