Another example of kinase domain truncation by alternative splicing is provided by the VEGFR (vascular endothelial growth factor (VEGF) Receptor) family, which encodes membrane-bound tyrosine kinase receptors for VEGFs acting as key regulators of vasculogenesis, angiogenesis, and lymphangiogenesis, both in physiological conditions and in cancer [30, 31]. This evidence concerns the gene NTRK1 and cancer.