Lung biopsies from patients afflicted with severe Idiopathic Pulmonary Fibrosis displayed a high level expression of both active, phosphorylated human C/EBPβ on Thr266 and phosphorylated RSK (that co-localized in activated α-SMA+ LMF within the fibrous tissue) (Figures 9 and S5), compared with samples from two matched control patients as identified by confocal scanning microscopy with specific antibodies against RSK-PhosphoSer380, C/EBPβ-PhosphoThr266, and α-SMA for LMF [1]. The gene discussed is ACTA1; the disease is pulmonary fibrosis.