NTHi infection alone did increase neutrophil influx into the airways (p<0.01), IL-17 mRNA expression in the lung (p<0.05) and the percentages of lung macrophages and neutrophils producing IL-17 (both p<0.001) 1 day after infection, compared to uninfected, nonallergic (Saline) controls. Here, IL17A is linked to infection.