IL17A and flatulence: Notably, however, a larger fraction of the 2W:I-Ab-specific T cell population produced IL-17A following intranasal GAS-2W infection than following intranasal LM-2W infection (Fig. 5B) [20] ; Furthermore, both GAS and LM primarily induced a Th1 response to intravenous inoculation but LM induced a significantly greater Th1 response than GAS (Fig. 5B).