CXCL2 and hydrops fetalis: Therefore, in the present study, either Leptin in BAL acted anti-inflammatory and prevented an increased particle related neutrophil influx in CA and HF mice, or, if BAL Leptin action was pro-inflamamtory, then other molecular factors may have counterbalanced, like the reduced MIP2 and IFNγ concentrations in BAL, which predominantly can be dedicated to be of alveolar macrophage origin.