Exonic deletions of 11p15.5 in Wilms' tumors [5] and loss of heterozygosity in hepatoblastomas [8] have also been reported, indicating that SLC22A18 may play a role in tumorigenesis; however, SLC22A18 expression in glioma and the relationship with glioma progression has not previously been investigated in humans. Here, SLC67A1 is linked to glioma.