We now have molecular biological data suggesting an epilepsy-related mechanism related to BRD2. Although the data are preliminary, we have observed that the ratio of normally-spliced to alternatively spliced (and non-functional) BRD2 RNA is a function of the number of tandem GT repeats in a microsatellite located in intron 2 of BRD2[25]. Here, BRD2 is linked to epilepsy.