AICDA and neoplasm: While a direct correlation between miR-93 activity and AID-induced oncogenesis remains to be described, miR-93 perturbations have been found to enhance cell survival, possess oncogenic activities, and augment tumor growth through regulating integrin-β8, the tumor suppressor gene FUS1, the Cdk inhibitor p21, and tumor protein 53-induced nuclear protein 1 (TP53INP1) [39-42].