IL10 and bronchiolitis: These findings, along with evidence of a link between a polymorphism in the IL-10 locus and the severity of bronchiolitis in infants infected with RSV [11], [12], [13], [14], prompted us to explore the role of IL-10, and in particular of IL-10 produced by virus-specific effector T-cells, in controlling pulmonary injury associated with experimental murine RSV infection.