IL17A and infection: Recently, in p47phox−/−mice a pathogenic link between defective NADPH oxidase, reduced tryptophan catabolism, and IL-17-mediated inflammation [35] has been shown.After infection with Aspergillus fumigatus, these mice develop an enormous inflammatory response with a shift in the γδ T cell system and overproduction of IL-17 in comparison to wild-type mice.