In addition, astrocyte activity, which is concomitant with motor neuron degeneration and normally increases at later stages of ALS disease [21,22], did not change between wild-type and MLC/SOD1G93A spinal cord, while it selectively increased in the SOD1G93A spinal cord at later stages of the disease as revealed by glial fibrillary acidic protein (GFAP) expression (Figure 2C). The gene discussed is GFAP; the disease is amyotrophic lateral sclerosis.